Although vascular injury after humeral fracture is very rare, it is a complication that has serious sequelae. It has been associated with proximal humeral fracture or shoulder dislocation in adults and humeral supracondylar fracture in children. However, delayed brachial artery occlusion after humeral shaft fracture has never been reported worldwide. Nevertheless, delayed brachial artery occlusion after humerus shaft fracture has the potential to cause serious complications in the short term as well as long term; therefore, it is essential to provide accurate diagnosis and prompt treatment. We report a case of delayed brachial artery occlusion after humeral shaft open fracture that was successfully treated with early diagnosis as well as effective treatment.
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Delayed presentation of brachial artery injury following fracture shaft humerus; whether amputate or salvage: A series of two cases Bhanu Sharma, Sibashish Metia, Kavish Kapoor, Pankaj Poswal Journal of Orthopedics, Traumatology and Rehabilitation.2018; 10(2): 137. CrossRef
Although vascular injury after clavicular fracture is a extremely rare, it is a complication which is serious problem. Vascular injury associated with the fracture can be immediate or delayed. We report a case of late-onset brachial artery occlusion caused by subclavian artery stenosis with excessive scar tissue after open reduction and plate fixation for clavicular fracture and include a review of the literature.
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Bilateral Brachial Artery Disease Presenting with Features of Raynaud’s Phenomenon: A Case Report and Review of the Literature Karan Seegobin, Brittany Lyons, Satish Maharaj, Cherisse Baldeo, Pramod Reddy, James Cunningham Case Reports in Vascular Medicine.2017; 2017: 1. CrossRef
Delayed Brachial Artery Occlusion after Humeral Shaft Open Fracture - A Case Report - Chul-Hyun Cho, Ki-Cheor Bae, Kyung-Jae Lee, Si-Wook Lee Journal of the Korean Fracture Society.2012; 25(2): 146. CrossRef
A 60 year old patient with a sudden thromboembolic occlusion of ipsilateral popliteal artery on four days after the total hip replacement (THR) were treated with high-dose urokinase by direct intraarterial selective infusion.
The cause of arterial occlusion after THR was not clear, but it was thought to be caused by spontaneous thrornboembolism in an elderly patient accompanied with diffuse arteriosclerosis, and this multifocal arteriosclerosis was caused not to perform the vein graft immediately.
The initial infusion therapy with 4,000 IU/min for 2 hours of urokinase was failed but the second trial with same doses of urokinase in another 2 hours was succeed with complete clot lysis.
Then 500,000 IU/24 hours of urokinase was infused again, and total 1,500,000 IU/28 hours was used in this patient. But massive internal bleeding from the operation site, hip joint, for more than 1,400 co was leaked because of bleeding tendency induced by extensive use of urokinase within short duration, and minor toe amputations should be performed on 2 weeks after thrombolysis because of distal migration of small thromboembolic particles.
It was suggested that the peripheral arterial occlusion resulting from thromboembolism after joint replacement, especially in an elderly patient with diffuse arteriosclerosis, could develop, and it could be successfully treated with an initially high-dose urokinase regimen if it is detected earlier, rather than vein graft or amputation.